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产品概述
产品简介
产品说明
产品形态 | 液体 |
产品浓度 | 1× |
产品规格 | 100mL/125mL×4 |
细菌检测 | 阴性 |
真菌检测 | 阴性 |
支原体检测 | 阴性 |
细胞生长实验 | 细胞生长良好,形态正常 |
内毒素含量(EU/mL) | ≤3 |
储存条件 | 2~8℃,避光储存 |
运输条件 | 冰袋冷藏运输 |
有效期 | 3个月 |
注意事项
参考文献
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EYE-503: A Novel Retinoic Acid Drug for Treating Retinal Neurodegeneration (2023-07-20)
作者:Sha Liu:1.The Affiliated Eye Hospital, Nanjing Medical University, Nanjing 210093, China. ; 2.The Fourth School of Clinical Medicine, Nanjing Medical University, Nanjing 210093, China. ; Yuke Ji:1.The Affiliated Eye Hospital, Nanjing Medical University, Nanjing 210093, China. ; 2.The Fourth School of Clinical Medicine, Nanjing Medical University, Nanjing 210093, China. ; Huan Li:1.The Affiliated Eye Hospital, Nanjing Medical University, Nanjing 210093, China. ; 2.The Fourth School of Clinical Medicine, Nanjing Medical University, Nanjing 210093, China. ; Ling Ren:1.Eye Institute and Department of Ophthalmology, Eye and ENT Hospital, State Key Laboratory of Medical Neurobiology, Fudan University, Shanghai 200433, China. ; Junya Zhu:1.The Affiliated Eye Hospital, Nanjing Medical University, Nanjing 210093, China. ; 2.The Fourth School of Clinical Medicine, Nanjing Medical University, Nanjing 210093, China. ; Tianjing Yang:1.The Affiliated Eye Hospital, Nanjing Medical University, Na
期刊:EYE-503: A Novel Retinoic Acid Drug for Treating Retinal Neurodegeneration
影响因子 :4.6
引用产品: 小鼠视网膜神经节细胞完全培养基
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Identification of circular RNA-Dcaf6 as a therapeutic target for optic nerve crush-induced RGC degeneration (2023-12-30)
作者:Yuke Ji:1.Department of Ophthalmology and Optometry, The Affiliated Eye Hospital, Nanjing Medical University, Nanjing, China; Department of Ophthalmology and Optometry, The Fourth School of Clinical Medicine, Nanjing Medical University, Nanjing, China. ; Jing Li:1.Eye Institute and Department of Ophthalmology, Eye and ENT Hospital, State Key Laboratory of Medical Neurobiology, Fudan University, Shanghai, China. ; Sha Liu:1.Department of Ophthalmology and Optometry, The Affiliated Eye Hospital, Nanjing Medical University, Nanjing, China; Department of Ophthalmology and Optometry, The Fourth School of Clinical Medicine, Nanjing Medical University, Nanjing, China. ; Junya Zhu:1.Department of Ophthalmology and Optometry, The Affiliated Eye Hospital, Nanjing Medical University, Nanjing, China; Department of Ophthalmology and Optometry, The Fourth School of Clinical Medicine, Nanjing Medical University, Nanjing, China. ; Jin Yao:1.Department of Ophthalmology and Optometry, The Affiliated
期刊:Identification of circular RNA-Dcaf6 as a therapeutic target for optic nerve crush-induced RGC degeneration
DOI:10.1016/j.ygeno.2023.110776
影响因子 :4.4
引用产品: 小鼠视网膜神经节细胞完全培养基
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Loss of pleckstrin homology domain and leucine-rich repeat protein phosphatase 2 has protective effects on high glucose-injured retinal ganglion cells via the effect on the Akt–GSK–3β–Nrf2 pathway (2022-12-23)
作者:Xuan Liu:1.Department of Ophthalmology, The First Affiliated Hospital of Xi'an Jiaotong University, No. 277 Yanta West Road, Xi'an, 710061, Shaanxi, China. liuxuan0831@xjtufh.edu.cn. ; Yong Liu:1.The Institute of Neurobiology, Xi'an Jiaotong University Health Science Center, No. 76 Yanta Road, Xi'an, 710061, Shaanxi, China. liuy5599@mail.xjtu.edu.cn. ; Li Chen:1.Department of Ophthalmology, The First Affiliated Hospital of Xi'an Jiaotong University, No. 277 Yanta West Road, Xi'an, 710061, Shaanxi, China. ; Zhichao Zhang:1.The Institute of Neurobiology, Xi'an Jiaotong University Health Science Center, No. 76 Yanta Road, Xi'an, 710061, Shaanxi, China. ; Lijun Cui:1.Department of Ophthalmology, The First Affiliated Hospital of Xi'an Jiaotong University, No. 277 Yanta West Road, Xi'an, 710061, Shaanxi, China. ; Ting Wei:1.Department of Ophthalmology, The First Affiliated Hospital of Xi'an Jiaotong University, No. 277 Yanta West Road, Xi'an, 710061, Shaanxi, China. ;
期刊:Loss of pleckstrin homology domain and leucine-rich repeat protein phosphatase 2 has protective effects on high glucose-injured retinal ganglion cells via the effect on the Akt–GSK–3β–Nrf2 pathway
DOI:10.1007/s00011-022-01680-1
影响因子 :7.0
引用产品: 小鼠视网膜神经节细胞 , 小鼠视网膜神经节细胞完全培养基
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A ZFP42/MARK2 regulatory network reduces the damage of retinal ganglion cells in glaucoma: a study based on GEO dataset and in vitro experiments (2022-09-21)
作者:Yuan Yin:1.Department of Ophthalmology, The Second Hospital of Jilin University, Changchun, 130042, Jilin, People's Republic of China. ; Shuai Wu:1.Department of Ophthalmology, The Second Hospital of Jilin University, Changchun, 130042, Jilin, People's Republic of China. ; Lingzhi Niu:1.Department of Ophthalmology, The First Affiliated Hospital of Shandong First Medical University & Shandong Provincial Qianfoshan Hospital, Jinan, 250014, People's Republic of China. ; Shiwei Huang:1.Department of Ophthalmology, The Second Hospital of Jilin University, Changchun, 130042, Jilin, People's Republic of China. hsw1979@jlu.edu.cn. ;
期刊:A ZFP42/MARK2 regulatory network reduces the damage of retinal ganglion cells in glaucoma: a study based on GEO dataset and in vitro experiments
DOI:10.1007/s10495-022-01746-9
影响因子 :5.6
引用产品: 小鼠视网膜神经节细胞 , 小鼠视网膜神经节细胞完全培养基
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Sulfiredoxin-1 protects retinal ganglion cells from high glucose-induced oxidative stress and inflammatory injury by potentiating Nrf2 signaling via the Akt/GSK-3β pathway (2010-01-20)
作者:Fei Zhu:1.Ophthalmology, Yulin Hospital of Traditional Chinese Medicine, Yulin 719000, China. ; Juan Shao:1.Ophthalmology, Shaanxi Eye Hospital, Xi'an People's Hospital (Xi'an Fourth Hospital), Affiliated Guangren Hospital, School of Medicine, Xi'an Jiaotong University, Xi'an 710004, China. Electronic address: shaojuan_sofia@163.com. ; Yunlin Tian:1.Ophthalmology, Shaanxi Eye Hospital, Xi'an People's Hospital (Xi'an Fourth Hospital), Affiliated Guangren Hospital, School of Medicine, Xi'an Jiaotong University, Xi'an 710004, China. ; Zhiguo Xu:1.Ophthalmology, Shaanxi Eye Hospital, Xi'an People's Hospital (Xi'an Fourth Hospital), Affiliated Guangren Hospital, School of Medicine, Xi'an Jiaotong University, Xi'an 710004, China. ;
期刊:Sulfiredoxin-1 protects retinal ganglion cells from high glucose-induced oxidative stress and inflammatory injury by potentiating Nrf2 signaling via the Akt/GSK-3β pathway
DOI:10.1016/j.intimp.2021.108221
影响因子 :5.7
引用产品: 小鼠视网膜神经节细胞完全培养基
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