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产品概述
产品简介
NCI-H295R细胞专用培养基由普诺赛技术团队精心优化，经过长期测试，本产品可保持NCI-H295R细胞最佳的生长状态。本产品中已包含NCI-H295R细胞生长所需的各种成分，无需添加任何成分，可直接用于NCI-H295R细胞的体外培养。本产品仅供进一步科研使用，不得用于诊断、治疗、临床、家庭及其它用途。
产品说明
| 产品形态 | 液体 |
| 产品浓度 | 1× |
| 产品规格 | 125mL×4 |
| 培养基成分 | DMEM/F12(PM150312)＋0.5% ITS-G（100×）(PB180429)＋10% FBS(164210-50)＋1% P/S(PB180120) |
| 细菌检测 | 阴性 |
| 真菌检测 | 阴性 |
| 支原体检测 | 阴性 |
| 细胞生长实验 | 细胞生长良好，形态正常 |
| 内毒素含量(EU/mL) | ≤3 |
| 储存条件 | 2~8℃，避光储存 |
| 运输条件 | 冰袋冷藏运输 |
| 有效期 | 3个月 |
注意事项
参考文献
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Phosphoproteomics Reveals the Wolframin-Calcium Axis as an Important Pathogenic Signaling Node in Primary Aldosteronism (2023-02-24)
作者：Linqiang Ma:1.Department of Endocrinology (L.M., S.Y., Y.Y., X.C., Y.H., Q.C., J.L., B.K., S.T., X.L., H.S., Y.J., Y.S., Q.L., J.H.), the First Affiliated Hospital of Chongqing Medical University, Chongqing, China. ; 2.The Chongqing Key Laboratory of Translational Medicine in Major Metabolic Diseases (L.M., S.Y., J.L., Q.L., J.H.), the First Affiliated Hospital of Chongqing Medical University, Chongqing, China. ; Shumin Yang:1.Department of Endocrinology (L.M., S.Y., Y.Y., X.C., Y.H., Q.C., J.L., B.K., S.T., X.L., H.S., Y.J., Y.S., Q.L., J.H.), the First Affiliated Hospital of Chongqing Medical University, Chongqing, China. ; 2.The Chongqing Key Laboratory of Translational Medicine in Major Metabolic Diseases (L.M., S.Y., J.L., Q.L., J.H.), the First Affiliated Hospital of Chongqing Medical University, Chongqing, China. ; Yi Yang:1.Department of Endocrinology (L.M., S.Y., Y.Y., X.C., Y.H., Q.C., J.L., B.K., S.T., X.L., H.S., Y.J., Y.S., Q.L., J.H.), the First Affiliated Hospital of C
期刊：Phosphoproteomics Reveals the Wolframin-Calcium Axis as an Important Pathogenic Signaling Node in Primary Aldosteronism
DOI：10.1161/HYPERTENSIONAHA.122.20515
影响因子 ：8.3
引用产品： NCI-H295R细胞专用培养基
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CDK1 serves as a therapeutic target of adrenocortical carcinoma via regulating epithelial–mesenchymal transition, G2/M phase transition, and PANoptosis (2022-10-02)
作者：Liwen Ren:1.The State Key Laboratory of Bioactive Substance and Function of Natural Medicines, Beijing, 100050, China. ; 2.Key Laboratory of Drug Target Research and Drug Screen, Institute of Materia Medica, Chinese Academy of Medical Science and Peking Union Medical College, Beijing, 100050, China. ; Yihui Yang:1.The State Key Laboratory of Bioactive Substance and Function of Natural Medicines, Beijing, 100050, China. ; 2.Key Laboratory of Drug Target Research and Drug Screen, Institute of Materia Medica, Chinese Academy of Medical Science and Peking Union Medical College, Beijing, 100050, China. ; Wan Li:1.The State Key Laboratory of Bioactive Substance and Function of Natural Medicines, Beijing, 100050, China. ; 2.Key Laboratory of Drug Target Research and Drug Screen, Institute of Materia Medica, Chinese Academy of Medical Science and Peking Union Medical College, Beijing, 100050, China. ; Xiangjin Zheng:1.The State Key Laboratory of Bioactive Substance and Function of Natural M
期刊：CDK1 serves as a therapeutic target of adrenocortical carcinoma via regulating epithelial–mesenchymal transition, G2/M phase transition, and PANoptosis
DOI：10.1186/s12967-022-03641-y
影响因子 ：8.4
引用产品： NCI-H295R 细胞 , SW-13 细胞 , SW-13细胞专用培养基 (L15) , NCI-H295R细胞专用培养基
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IFNγ enhances ferroptosis by increasing JAK‑STAT pathway activation to suppress SLCA711 expression in adrenocortical carcinoma (2003-02-20)
作者：Xinbo Yu:1.Department of Urology, Ruijin Hospital, Shanghai Jiaotong University School of Medicine, Shanghai 200025, P.R. China. ; Dandan Zhu:1.Department of Urology, Ruijin Hospital, Shanghai Jiaotong University School of Medicine, Shanghai 200025, P.R. China. ; Bixian Luo:1.Department of General Surgery, Ruijin Hospital, Shanghai Jiaotong University School of Medicine, Shanghai 200025, P.R. China. ; Wei Kou:1.Department of Urology, Ruijin Hospital, Shanghai Jiaotong University School of Medicine, Shanghai 200025, P.R. China. ; Yuling Cheng:1.Department of Urology, Ruijin Hospital, Shanghai Jiaotong University School of Medicine, Shanghai 200025, P.R. China. ; Yu Zhu:1.Department of Urology, Ruijin Hospital, Shanghai Jiaotong University School of Medicine, Shanghai 200025, P.R. China. ;
期刊：IFNγ enhances ferroptosis by increasing JAK‑STAT pathway activation to suppress SLCA711 expression in adrenocortical carcinoma
影响因子 ：4.2
引用产品： NCI-H295R 细胞 , NCI-H295R细胞专用培养基
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