Hs 578T (人乳腺癌细胞) (STR鉴定正确)

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参考文献

• Vertical pathway inhibition of receptor tyrosine kinases and BAD with synergistic efficacy in triple negative breast cancer (2024-01-10)

  作者：Yan Qin Tan:1.Institute of Biopharmaceutical and Health Engineering, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, 518055, People's Republic of China. ; 2.Tsinghua Berkeley Shenzhen Institute, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, 518055, Guangdong, People's Republic of China. ; Yi-Shiou Chiou:1.Tsinghua Berkeley Shenzhen Institute, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, 518055, Guangdong, People's Republic of China. ; 2.Master Degree Program in Toxicology, College of Pharmacy, Kaohsiung Medical University, Kaohsiung, 807, Taiwan. ; 3.Shenzhen Bay Laboratory, Shenzhen, 518055, Guangdong, People's Republic of China. ; 4.Department of Medical Research, Kaohsiung Medical University Hospital, Kaohsiung, 807, Taiwan. ; Hui Guo:1.Tsinghua Berkeley Shenzhen Institute, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, 518055, Guangdong, People's R

  期刊：Vertical pathway inhibition of receptor tyrosine kinases and BAD with synergistic efficacy in triple negative breast cancer

  DOI：10.1038/s41698-023-00489-3

  影响因子 ：7.9

  引用产品： MDA-MB-231/GFP 细胞 ,  BT-549 细胞 ,  HCC1937 细胞 ,  Hs 578T 细胞

• The dePARylase NUDT16 promotes radiation resistance of cancer cells by blocking SETD3 for degradation via reversing its ADP-ribosylation (2024-01-22)

  作者：Weijun Wu:1.Guangdong Provincial Key Laboratory of Malignant Tumor Epigenetics and Gene Regulation, Guangdong-Hong Kong Joint Laboratory for RNA Medicine, Medical Research Center, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, China; Department of Oncology Radiotherapy, The First Affiliated Hospital, Hengyang Medical School, University of South China, Hengyang, Hunan, China. ; Wenjing Wu:1.Guangdong Provincial Key Laboratory of Malignant Tumor Epigenetics and Gene Regulation, Guangdong-Hong Kong Joint Laboratory for RNA Medicine, Medical Research Center, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, China; Department of Breast Oncology, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, China. ; Yingshi Zhou:1.Guangdong Provincial Key Laboratory of Malignant Tumor Epigenetics and Gene Regulation, Guangdong-Hong Kong Joint Laboratory for RNA Medicine, Medical Research Center, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, G

  期刊：The dePARylase NUDT16 promotes radiation resistance of cancer cells by blocking SETD3 for degradation via reversing its ADP-ribosylation

  DOI：10.1016/j.jbc.2024.105671

  影响因子 ：4.8

  引用产品： MCF 10A 细胞 ,  HCC1806 细胞 ,  HCC1937 细胞 ,  MDA-MB-468 细胞 ,  BT-20 细胞 ,  Hs 578T 细胞 ,  MDA-MB-436 细胞 ,  MCF 10A细胞专用培养基 ,  HCC1806细胞专用培养基 ,  HCC1937细胞专用培养基 ,  MDA-MB-468细胞专用培养基 (L15) ,  BT-20细胞专用培养基 ,  Hs 578T细胞专用培养基 ,  MDA-MB-436细胞专用培养基 (L15)

• A novel polypeptide CAPG-171aa encoded by circCAPG plays a critical role in triple-negative breast cancer (2023-07-05)

  作者：Runjie Song:1.State Key Laboratory of Agrobiotechnology, College of Biological Sciences, China Agricultural University, Beijing, 100193, China. ; Peilan Guo:1.State Key Laboratory of Agrobiotechnology, College of Biological Sciences, China Agricultural University, Beijing, 100193, China. ; Xin Ren:1.State Key Laboratory of Agrobiotechnology, College of Biological Sciences, China Agricultural University, Beijing, 100193, China. ; Lijun Zhou:1.State Key Laboratory of Agrobiotechnology, College of Biological Sciences, China Agricultural University, Beijing, 100193, China. ; Peng Li:1.State Key Laboratory of Agrobiotechnology, College of Biological Sciences, China Agricultural University, Beijing, 100193, China. ; Nafis A Rahman:1.Department of Physiology, Institute of Biomedicine, University of Turku, Turku, Finland. ; 2.Department of Reproduction and Gynecological Endocrinology, Medical University of Bialystok, Bialystok, Poland. ; Sławomir Wołczyński:1.Department of Reproduction a

  期刊：A novel polypeptide CAPG-171aa encoded by circCAPG plays a critical role in triple-negative breast cancer

  DOI：10.1186/s12943-023-01806-x

  影响因子 ：37.3

  引用产品： MDA-MB-231 细胞 ,  MDA-MB-468 细胞 ,  293T [HEK-293T] 细胞 ,  Hs 578T 细胞 ,  MCF7 [MCF-7] 细胞 ,  T-47D 细胞 ,  ZR-75-1 细胞 ,  MCF 10A 细胞

• Icariin Induces Triple-Negative Breast Cancer Cell Apoptosis and Suppresses Invasion by Inhibiting the JNK/c-Jun Signaling Pathway (2023-03-28)

  作者：Shenghan Gao:1.The College of Life Sciences, Northwest University, Xi'an, 710069 People's Republic of China. ; 2.Department of Thyroid Breast Surgery, Xi'an NO.3 Hospital, the Affiliated Hospital of Northwest University, Xi'an, People's Republic of China. ; Xinyu Zhang:1.The College of Life Sciences, Northwest University, Xi'an, 710069 People's Republic of China. ; 2.Department of Thyroid Breast Surgery, Xi'an NO.3 Hospital, the Affiliated Hospital of Northwest University, Xi'an, People's Republic of China. ; Jie Liu:1.Clinical Medical Center, Xi'an NO.3 Hospital, the Affiliated Hospital of Northwest University, Xi'an, People's Republic of China. ; Fuqing Ji:1.Department of Thyroid Breast Surgery, Xi'an NO.3 Hospital, the Affiliated Hospital of Northwest University, Xi'an, People's Republic of China. ; Zhihao Zhang:1.Department of Thyroid Breast Surgery, Xi'an NO.3 Hospital, the Affiliated Hospital of Northwest University, Xi'an, People's Republic of China. ; Qingjie Meng:1.Depart

  期刊：Icariin Induces Triple-Negative Breast Cancer Cell Apoptosis and Suppresses Invasion by Inhibiting the JNK/c-Jun Signaling Pathway

  DOI：10.2147/DDDT.S398887

  影响因子 ：4.8

  引用产品： Hs 578T 细胞 ,  MDA-MB-468 细胞 ,  MCF 10A 细胞 ,  Hs 578T细胞专用培养基 ,  MDA-MB-468细胞专用培养基 (DMEM) ,  MCF 10A细胞专用培养基

• Gankyrin inhibits ferroptosis through the p53/SLC7A11/GPX4 axis in triple-negative breast cancer cells (2023-12-08)

  作者：Ming Lei:1.Department of Breast and Thyroid Surgery, The Second Affiliated Hospital, Hainan Medical University, Haikou, 570311, China. ; 2.Key Laboratory of Tropical Translational Medicine of Ministry of Education & School of Tropical Medicine, Hainan Medical University, Haikou, 571199, China. ; Yun-Long Zhang:1.Key Laboratory of Tropical Translational Medicine of Ministry of Education & School of Tropical Medicine, Hainan Medical University, Haikou, 571199, China. ; Feng-Ying Huang:1.Key Laboratory of Tropical Translational Medicine of Ministry of Education & School of Tropical Medicine, Hainan Medical University, Haikou, 571199, China. ; Heng-Yu Chen:1.Department of Breast and Thyroid Surgery, The Second Affiliated Hospital, Hainan Medical University, Haikou, 570311, China. ; Ming-Hui Chen:1.Key Laboratory of Tropical Translational Medicine of Ministry of Education & School of Tropical Medicine, Hainan Medical University, Haikou, 571199, China. ; Ri-Hong Wu:1.Key Laboratory of T

  期刊：Gankyrin inhibits ferroptosis through the p53/SLC7A11/GPX4 axis in triple-negative breast cancer cells

  DOI：10.1038/s41598-023-49136-8

  影响因子 ：4.6

  引用产品： MDA-MB-231 细胞 ,  HCC1937 细胞 ,  MDA-MB-468 细胞 ,  BT-20 细胞 ,  Hs 578T 细胞 ,  MCF 10A 细胞

• Elevated expression of the rhythm gene NFIL3 promotes the progression of TNBC by activating NF-κB signaling through suppression of NFKBIA transcription (2002-02-20)

  作者：Weiwei Yang:1.Department of Pathology, Harbin Medical University, Harbin, China. ; Jing Li:1.Department of Pathology, Harbin Medical University, Harbin, China. ; 2.Electronic Microscope Center of Harbin Medical University, Harbin, China. ; Minghui Zhang:1.Department of Oncology, Chifeng City Hospital, Chifeng, China. ; Haichuan Yu:1.School of Medical Laboratory, Xinxiang Medical University, Xinxiang, China. ; Yuan Zhuang:1.Department of Pathology, Harbin Medical University, Harbin, China. ; Lingyu Zhao:1.Department of Pathology, Harbin Medical University, Harbin, China. ; Lili Ren:1.Department of Pathology, Harbin Medical University, Harbin, China. ; Jinan Gong:1.Clinicopathological Diagnosis Center, Qiqihar Medical University, Qiqihar, China. ; Hongjie Bi:1.Department of Pathology, Harbin Medical University, Harbin, China. ; Lixuan Zeng:1.Department of Pathology, Harbin Medical University, Harbin, China. ; Yang Xue:1.Department of Pathology, Harbin Medical University, Harbin

  期刊：Elevated expression of the rhythm gene NFIL3 promotes the progression of TNBC by activating NF-κB signaling through suppression of NFKBIA transcription

  DOI：10.1186/s13046-022-02260-1

  影响因子 ：11.3

  引用产品： Hs 578T 细胞 ,  BT-549 细胞 ,  HCT-15 细胞 ,  MCF 10A 细胞 ,  A549 [A-549] 细胞 ,  SK-OV-3 细胞 ,  Hela 细胞 ,  PANC-1 细胞

• The inhibition of centromere protein K causes anticancer effects in breast carcinoma via effects on the FAK/PI3K/AKT/mTOR pathway (2022-09-09)

  作者：Jiao Yu:1.Department of Radiation Oncology, Shaanxi Provincial People's Hospital, Xi'an 710068, China. ; Kai Wang:1.Department of Traditional Chinese Medicine, Xi'an Chang'an District Hospital, Xi'an 710119, China. ; Sanhu Yang:1.Department of Thoracic Surgery, Tangdu Hospital, Air Force Military Medical University, Xi'an 710049, China. ; Gang Li:1.Department of Thyroid and Breast Surgery, Xi'an Daxing Hospital, Xi'an 710068, China. Electronic address: dooopenitlg@163.com. ;

  期刊：The inhibition of centromere protein K causes anticancer effects in breast carcinoma via effects on the FAK/PI3K/AKT/mTOR pathway

  DOI：10.1016/j.taap.2022.116232

  影响因子 ：4.5

  引用产品： T-47D 细胞 ,  Hs 578T 细胞 ,  MCF7 [MCF-7] 细胞 ,  BT-20 细胞 ,  BT-549 细胞 ,  MCF 10A 细胞 ,  RPMI-1640 培养基 ,  MEM 培养基 ,  DMEM/F12 培养基

• ISLR affects colon cancer progression by regulating the epithelial–mesenchymal transition signaling pathway (2001-02-20)

  作者：Chunhua Chi:1.Department of Anorectal Surgery, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan. ; Tongming Liu:1.Department of Anorectal Surgery, Feicheng People's hospital, Tai An. ; Shengnan Yang:1.Department of Proctology, Changqing District Hospital of Traditional Chinese Medicine, Jinan, Shandong, P.R. China. ; Benjun Wang:1.Department of Anorectal Surgery, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan. ; Weiwei Han:1.Department of Anorectal Surgery, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan. ; Jiansheng Li:1.Department of Anorectal Surgery, Affiliated Hospital of Shandong University of Traditional Chinese Medicine, Jinan. ;

  期刊：ISLR affects colon cancer progression by regulating the epithelial–mesenchymal transition signaling pathway

  DOI：10.1097/CAD.0000000000001233

  影响因子 ：2.3

  引用产品： 人结肠黏膜上皮细胞 ,  CX-1 细胞 ,  SW480 [SW-480] 细胞 ,  HCT 116 细胞 ,  HT-29 细胞 ,  Hs 578T 细胞

• MCPIP1-mediated NFIC alternative splicing inhibits proliferation of triple-negative breast cancer via cyclin D1-Rb-E2F1 axis (2004-01-20)

  作者：Fengxia Chen:1.Hubei Key Laboratory of Tumor Biological Behaviors, Zhongnan Hospital of Wuhan University, Wuhan, China. ; 2.Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China. ; 3.Hubei Cancer Clinical Study Center, Zhongnan Hospital of Wuhan University, Wuhan, China. ; Qingqing Wang:1.Hubei Key Laboratory of Tumor Biological Behaviors, Zhongnan Hospital of Wuhan University, Wuhan, China. ; 2.Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China. ; 3.Hubei Cancer Clinical Study Center, Zhongnan Hospital of Wuhan University, Wuhan, China. ; Xiaoyan Yu:1.Hubei Key Laboratory of Tumor Biological Behaviors, Zhongnan Hospital of Wuhan University, Wuhan, China. ; 2.Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, China. ; 3.Hubei Cancer Clinical Study Center, Zhongnan Hospital of Wuhan University, Wuhan, China. ; Ningning Yang:1.Hubei Key Laboratory of Tumo

  期刊：MCPIP1-mediated NFIC alternative splicing inhibits proliferation of triple-negative breast cancer via cyclin D1-Rb-E2F1 axis

  DOI：10.1038/s41419-021-03661-4

  影响因子 ：9.0

  引用产品： MDA-MB-157 细胞 ,  Hs 578T 细胞 ,  BT-549 细胞 ,  MDA-MB-231 细胞 ,  MDA-MB-468 细胞 ,  MCF 10A 细胞 ,  293T [HEK-293T] 细胞 ,  DMEM/F12完全培养基 (含10%FBS)

• Fat mass and obesity-associated protein (FTO) mediates signal transducer and activator of transcription 3 (STAT3)-drived resistance of breast cancer to doxorubicin (2006-01-20)

  作者：Yan Wang:1.Department of Pathology, Shenzhen People's Hospital, The Second Clinical Medical College of Jinan University, the First Affiliated Hospital of Southern University of Science and Technology, Shenzhen, China. ; Zhiqiang Cheng:1.Department of Pathology, Shenzhen People's Hospital, The Second Clinical Medical College of Jinan University, the First Affiliated Hospital of Southern University of Science and Technology, Shenzhen, China. ; Jing Xu:1.Department of Pathology, Shenzhen People's Hospital, The Second Clinical Medical College of Jinan University, the First Affiliated Hospital of Southern University of Science and Technology, Shenzhen, China. ; Meina Lai:1.Department of Pathology, Shenzhen People's Hospital, The Second Clinical Medical College of Jinan University, the First Affiliated Hospital of Southern University of Science and Technology, Shenzhen, China. ; Liming Liu:1.Department of Pathology, Shenzhen People's Hospital, The Second Clinical Medical College of Jinan

  期刊：Fat mass and obesity-associated protein (FTO) mediates signal transducer and activator of transcription 3 (STAT3)-drived resistance of breast cancer to doxorubicin

  DOI：10.1080/21655979.2021.1924544

  影响因子 ：4.9

  引用产品： MCF7 [MCF-7] 细胞 ,  MDA-MB-231 细胞 ,  Hs 578T 细胞 ,  Leibovitz's L-15 培养基

• MiR‐96‐5p promotes breast cancer migration by activating MEK/ERK signaling (2002-00-20)

  作者：Wei-Yan Qin:1.Department of Surgery, The Second Affiliated Hospital of Soochow University, China. ; 2.Department of General Surgery, The First People's Hospital of Nantong, China. ; Shi-Chun Feng:1.Department of General Surgery, The First People's Hospital of Nantong, China. ; Yong-Qiang Sun:1.Department of General Surgery, The First People's Hospital of Nantong, China. ; Guo-Qin Jiang:1.Department of Surgery, The Second Affiliated Hospital of Soochow University, China. ;

  期刊：MiR‐96‐5p promotes breast cancer migration by activating MEK/ERK signaling

  DOI：10.1002/jgm.3188

  影响因子 ：3.5

  引用产品： BT-549 细胞 ,  Hs 578T 细胞 ,  MDA-MB-231 细胞 ,  MCF 10A 细胞

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