HCC1937 (人乳腺癌细胞) (STR鉴定正确)

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参考文献

• Inhibition of ATM promotes PD-L1 expression by activating JNK/c-Jun/TNF-α signaling axis in triple-negative breast cancer (2024-01-24)

  作者：Chenying Liu:1.Department of Breast Cancer Pathology and Research Laboratory, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Center for Cancer, Key Laboratory of Breast Cancer Prevention and Therapy, Tianjin's Clinical Research Center for Cancer, Tianjin, 300060, China. ; Xiaolong Qian:1.Department of Breast Cancer Pathology and Research Laboratory, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Center for Cancer, Key Laboratory of Breast Cancer Prevention and Therapy, Tianjin's Clinical Research Center for Cancer, Tianjin, 300060, China. ; Chunyan Yu:1.Tianjin Institute of Immunology, Department of Immunology, Tianjin Key Laboratory of Cellular and Molecular Immunology, Key Laboratory of Diseases and Microenvironment of Ministry of Education of China, Tianjin Medical University, Tianjin, 300070, China. ; Xiaoqing Xia:1.Department of Breast Cancer Pathology and Research Laboratory, Tianjin Medical University

  期刊：Inhibition of ATM promotes PD-L1 expression by activating JNK/c-Jun/TNF-α signaling axis in triple-negative breast cancer

  DOI：10.1016/j.canlet.2024.216642

  影响因子 ：9.7

  引用产品： MDA-MB-231 细胞 ,  HCC1937 细胞

• Vertical pathway inhibition of receptor tyrosine kinases and BAD with synergistic efficacy in triple negative breast cancer (2024-01-10)

  作者：Yan Qin Tan:1.Institute of Biopharmaceutical and Health Engineering, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, 518055, People's Republic of China. ; 2.Tsinghua Berkeley Shenzhen Institute, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, 518055, Guangdong, People's Republic of China. ; Yi-Shiou Chiou:1.Tsinghua Berkeley Shenzhen Institute, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, 518055, Guangdong, People's Republic of China. ; 2.Master Degree Program in Toxicology, College of Pharmacy, Kaohsiung Medical University, Kaohsiung, 807, Taiwan. ; 3.Shenzhen Bay Laboratory, Shenzhen, 518055, Guangdong, People's Republic of China. ; 4.Department of Medical Research, Kaohsiung Medical University Hospital, Kaohsiung, 807, Taiwan. ; Hui Guo:1.Tsinghua Berkeley Shenzhen Institute, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, 518055, Guangdong, People's R

  期刊：Vertical pathway inhibition of receptor tyrosine kinases and BAD with synergistic efficacy in triple negative breast cancer

  DOI：10.1038/s41698-023-00489-3

  影响因子 ：7.9

  引用产品： MDA-MB-231/GFP 细胞 ,  BT-549 细胞 ,  HCC1937 细胞 ,  Hs 578T 细胞

• Triple-Negative Breast Cancer Intrinsic FTSJ1 Favors Tumor Progression and Attenuates CD8+ T Cell Infiltration (2024-01-31)

  作者：Yangqing Sun:1.Department of Oncology, Xiangya Hospital, Central South University, Changsha 410008, China. ; Qingqing Liu:1.Department of Oncology, Xiangya Hospital, Central South University, Changsha 410008, China. ; Shangwei Zhong:1.The Cancer Research Institute and the Second Affiliated Hospital, Hengyang Medical School, University of South China, Hengyang 421001, China. ; Rui Wei:1.Department of Oncology, Xiangya Hospital, Central South University, Changsha 410008, China. ; Jun-Li Luo:1.Department of Oncology, Xiangya Hospital, Central South University, Changsha 410008, China. ; 2.The Cancer Research Institute and the Second Affiliated Hospital, Hengyang Medical School, University of South China, Hengyang 421001, China. ; 3.National Health Commission Key Laboratory of Birth Defect Research and Prevention, Hunan Provincial Maternal and Child Health Care Hospital, Changsha 410008, China. ;

  期刊：Triple-Negative Breast Cancer Intrinsic FTSJ1 Favors Tumor Progression and Attenuates CD8+ T Cell Infiltration

  DOI：10.3390/cancers16030597

  影响因子 ：5.2

  引用产品： MDA-MB-231 细胞 ,  HCC1937 细胞 ,  4T1 细胞 ,  特级胎牛血清 ,  DMEM高糖 培养基 ,  RPMI-1640 培养基

• The dePARylase NUDT16 promotes radiation resistance of cancer cells by blocking SETD3 for degradation via reversing its ADP-ribosylation (2024-01-22)

  作者：Weijun Wu:1.Guangdong Provincial Key Laboratory of Malignant Tumor Epigenetics and Gene Regulation, Guangdong-Hong Kong Joint Laboratory for RNA Medicine, Medical Research Center, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, China; Department of Oncology Radiotherapy, The First Affiliated Hospital, Hengyang Medical School, University of South China, Hengyang, Hunan, China. ; Wenjing Wu:1.Guangdong Provincial Key Laboratory of Malignant Tumor Epigenetics and Gene Regulation, Guangdong-Hong Kong Joint Laboratory for RNA Medicine, Medical Research Center, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, China; Department of Breast Oncology, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, China. ; Yingshi Zhou:1.Guangdong Provincial Key Laboratory of Malignant Tumor Epigenetics and Gene Regulation, Guangdong-Hong Kong Joint Laboratory for RNA Medicine, Medical Research Center, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, G

  期刊：The dePARylase NUDT16 promotes radiation resistance of cancer cells by blocking SETD3 for degradation via reversing its ADP-ribosylation

  DOI：10.1016/j.jbc.2024.105671

  影响因子 ：4.8

  引用产品： MCF 10A 细胞 ,  HCC1806 细胞 ,  HCC1937 细胞 ,  MDA-MB-468 细胞 ,  BT-20 细胞 ,  Hs 578T 细胞 ,  MDA-MB-436 细胞 ,  MCF 10A细胞专用培养基 ,  HCC1806细胞专用培养基 ,  HCC1937细胞专用培养基 ,  MDA-MB-468细胞专用培养基 (L15) ,  BT-20细胞专用培养基 ,  Hs 578T细胞专用培养基 ,  MDA-MB-436细胞专用培养基 (L15)

• Discovery of Novel 4-Hydroxyquinazoline Derivatives: In Silico, In Vivo and In Vitro Studies Using Primary PARPi-Resistant Cell Lines (2024-03-21)

  作者：Lijie Zhu:1.School of Pharmacy, Shandong Second Medical University, Weifang 261053, China. ; Binzhuo Liu:1.School of Pharmacy, Shandong Second Medical University, Weifang 261053, China. ; Feng Jin:1.School of Pharmacy, Shandong Second Medical University, Weifang 261053, China. ; Weilong Cao:1.School of Pharmacy, Shandong Second Medical University, Weifang 261053, China. ; Guangzhao Xu:1.School of Pharmacy, Shandong Second Medical University, Weifang 261053, China. ; Xinwei Zhang:1.School of Pharmacy, Shandong Second Medical University, Weifang 261053, China. ; Peng Peng:1.School of Pharmacy (Preparatory), East China Normal University, Shanghai 200241, China. ; Dingding Gao:1.The Research Center of Chiral Drugs, Innovation Research Institute of Traditional Chinese Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China. ; Bin Wang:1.School of Pharmacy, Shandong Second Medical University, Weifang 261053, China. ; Kairui Feng:1.School of Pharmacy, Sh

  期刊：Discovery of Novel 4-Hydroxyquinazoline Derivatives: In Silico, In Vivo and In Vitro Studies Using Primary PARPi-Resistant Cell Lines

  DOI：10.3390/molecules29061407

  影响因子 ：4.6

  引用产品： HCT-15 细胞 ,  HCC1937 细胞 ,  MDA-MB-231 细胞 ,  HL-7702 [L-02; LO2] 细胞 ,  NCM460 细胞

• Oxypalmatine regulates proliferation and apoptosis of breast cancer cells by inhibiting PI3K/AKT signaling and its efficacy against breast cancer organoids (2023-03-10)

  作者：Xian Lin:1.Peking University Shenzhen Hospital; Shenzhen Key Laboratory of Inflammatory and Immunology Diseases, Shenzhen 518036, Guangdong, China. ; Dong Chen:1.Department of Thyroid and Breast Surgery, Peking University Shenzhen Hospital, Shenzhen 518036, Guangdong, China. ; Xinyu Chu:1.Department of Thyroid and Breast Surgery, Peking University Shenzhen Hospital, Shenzhen 518036, Guangdong, China. ; Lianxiang Luo:1.The Marine Biomedical Research Institute, Guangdong Medical University, Zhanjiang 524023, Guangdong, China. ; Zhihao Liu:1.The First Clinical College, Guangdong Medical University, Zhanjiang 524023, Guangdong, China. ; Jian Chen:1.Peking University Shenzhen Hospital; Shenzhen Key Laboratory of Inflammatory and Immunology Diseases, Shenzhen 518036, Guangdong, China. Electronic address: chenjian@pkuszh.com. ;

  期刊：Oxypalmatine regulates proliferation and apoptosis of breast cancer cells by inhibiting PI3K/AKT signaling and its efficacy against breast cancer organoids

  DOI：10.1016/j.phymed.2023.154752

  影响因子 ：7.9

  引用产品： MCF7 [MCF-7] 细胞 ,  HCC1937 细胞 ,  DU4475 细胞 ,  MDA-MB-231 细胞

• Metabolome Sequencing Reveals that Protein Arginine-N-Methyltransferase 1 Promotes the Progression of Invasive Micropapillary Carcinoma of the Breast and Predicts a Poor Prognosis (2023-06-09)

  作者：Kailiang Wu:1.Department of Breast Cancer Pathology and Research Laboratory, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Center for Cancer, Key Laboratory of Breast Cancer Prevention and Therapy, Tianjin's Clinical Research Center for Cancer, Tianjin, China; Department of Clinical Laboratory, Tianjin Medical University General Hospital, Tianjin, China. ; Weidong Li:1.Department of Breast Cancer Pathology and Research Laboratory, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Center for Cancer, Key Laboratory of Breast Cancer Prevention and Therapy, Tianjin's Clinical Research Center for Cancer, Tianjin, China. ; Hanjiao Liu:1.Department of Breast Cancer Pathology and Research Laboratory, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research Center for Cancer, Key Laboratory of Breast Cancer Prevention and Therapy, Tianjin's Clinical Research Center for Cancer, Tianjin, China.

  期刊：Metabolome Sequencing Reveals that Protein Arginine-N-Methyltransferase 1 Promotes the Progression of Invasive Micropapillary Carcinoma of the Breast and Predicts a Poor Prognosis

  DOI：10.1016/j.ajpath.2023.05.010

  影响因子 ：6.0

  引用产品： HCC1937 细胞 ,  MDA-MB-231 细胞 ,  293T [HEK-293T] 细胞

• Combined Inhibition of UBE2C and PLK1 Reduce Cell Proliferation and Arrest Cell Cycle by Affecting ACLY in Pan-Cancer (2023-10-27)

  作者：Keying Liang:1.School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China. ; Qian Wang:1.School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China. ; Li Qiu:1.School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China. ; Xiaocheng Gong:1.School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China. ; Zixi Chen:1.School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China. ; Haibo Zhang:1.School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China. ; Ke Ding:1.School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China. ; Yunfei Liu:1.School of Biology and Biological Engineering, South China University of Technology, Guangzhou 510006, China. ; Ji

  期刊：Combined Inhibition of UBE2C and PLK1 Reduce Cell Proliferation and Arrest Cell Cycle by Affecting ACLY in Pan-Cancer

  DOI：10.3390/ijms242115658

  影响因子 ：5.6

  引用产品： HCC1937 细胞 ,  NCI-H520 细胞 ,  SNU-387 细胞 ,  SNU-182 细胞 ,  BEAS-2B 细胞 ,  MCF 10A细胞专用培养基

• Leveraging senescence-oxidative stress co-relation to predict prognosis and drug sensitivity in breast invasive carcinoma (2023-08-04)

  作者：Yinghui Ye:1.Department of Laboratory Medicine, Peking University Shenzhen Hospital, Shenzhen, China. ; Yulou Luo:1.Department of Breast Surgery, Affiliated Tumor Hospital of Xinjiang Medical University, Urumqi, China. ; Tong Guo:1.Department of Laboratory Medicine, Peking University Shenzhen Hospital, Shenzhen, China. ; Chenguang Zhang:1.Department of Breast Surgery, Affiliated Tumor Hospital of Xinjiang Medical University, Urumqi, China. ; Yutian Sun:1.Department of Medical Oncology, Sichuan Cancer Hospital and Institute, Sichuan Cancer Center, School of Medicine, University of Electronic Science and Technology of China, Chengdu, China. ; Anping Xu:1.Department of Laboratory Medicine, Peking University Shenzhen Hospital, Shenzhen, China. ; Ling Ji:1.Department of Laboratory Medicine, Peking University Shenzhen Hospital, Shenzhen, China. ; Jianghua Ou:1.Department of Breast Surgery, Affiliated Tumor Hospital of Xinjiang Medical University, Urumqi, China. ; Shang Ying Wu:1.Depa

  期刊：Leveraging senescence-oxidative stress co-relation to predict prognosis and drug sensitivity in breast invasive carcinoma

  DOI：10.3389/fendo.2023.1179050

  影响因子 ：5.2

  引用产品： MCF7 [MCF-7] 细胞 ,  BT-20 细胞 ,  MDA-MB-231 细胞 ,  HCC1806 细胞 ,  HCC1937 细胞 ,  MCF7 [MCF-7]细胞专用培养基 ,  BT-20细胞专用培养基 ,  MDA-MB-231细胞专用培养基 (L15) ,  HCC1806细胞专用培养基 ,  HCC1937细胞专用培养基

• Gankyrin inhibits ferroptosis through the p53/SLC7A11/GPX4 axis in triple-negative breast cancer cells (2023-12-08)

  作者：Ming Lei:1.Department of Breast and Thyroid Surgery, The Second Affiliated Hospital, Hainan Medical University, Haikou, 570311, China. ; 2.Key Laboratory of Tropical Translational Medicine of Ministry of Education & School of Tropical Medicine, Hainan Medical University, Haikou, 571199, China. ; Yun-Long Zhang:1.Key Laboratory of Tropical Translational Medicine of Ministry of Education & School of Tropical Medicine, Hainan Medical University, Haikou, 571199, China. ; Feng-Ying Huang:1.Key Laboratory of Tropical Translational Medicine of Ministry of Education & School of Tropical Medicine, Hainan Medical University, Haikou, 571199, China. ; Heng-Yu Chen:1.Department of Breast and Thyroid Surgery, The Second Affiliated Hospital, Hainan Medical University, Haikou, 570311, China. ; Ming-Hui Chen:1.Key Laboratory of Tropical Translational Medicine of Ministry of Education & School of Tropical Medicine, Hainan Medical University, Haikou, 571199, China. ; Ri-Hong Wu:1.Key Laboratory of T

  期刊：Gankyrin inhibits ferroptosis through the p53/SLC7A11/GPX4 axis in triple-negative breast cancer cells

  DOI：10.1038/s41598-023-49136-8

  影响因子 ：4.6

  引用产品： MDA-MB-231 细胞 ,  HCC1937 细胞 ,  MDA-MB-468 细胞 ,  BT-20 细胞 ,  Hs 578T 细胞 ,  MCF 10A 细胞

• Isopimaric acid, an ion channel regulator, regulates calcium and oxidative phosphorylation pathways to inhibit breast cancer proliferation and metastasis (2023-02-06)

  作者：Jiacheng Li:1.College of Life Science, Sichuan Normal University, Chengdu, Sichuan 610101, China; Zhejiang Provincial Key Laboratory of Cancer Prevention and Treatment Technology of Integrated Traditional Chinese and Western Medicine, Zhejiang Academy of Traditional Chinese Medicine, Tongde Hospital of Zhejiang Province, Hangzhou, Zhejiang, 310012, China. ; Xiaozhen Liu:1.Department of Medical and Radiation Oncology, Linyi People's Hospital, Linyi 276000, China. ; Lin Chen:1.Sericultural Research Institute, Zhejiang, Academy of Agricultural Sciences, Hangzhou 310021, China. ; Xinping Zhu:1.Zhejiang Provincial Key Laboratory of Cancer Prevention and Treatment Technology of Integrated Traditional Chinese and Western Medicine, Zhejiang Academy of Traditional Chinese Medicine, Tongde Hospital of Zhejiang Province, Hangzhou, Zhejiang, 310012, China. ; Zhihong Yu:1.Zhejiang Provincial Key Laboratory of Cancer Prevention and Treatment Technology of Integrated Traditional Chinese and Weste

  期刊：Isopimaric acid, an ion channel regulator, regulates calcium and oxidative phosphorylation pathways to inhibit breast cancer proliferation and metastasis

  DOI：10.1016/j.taap.2023.116415

  影响因子 ：3.8

  引用产品： MDA-MB-231 细胞 ,  MCF7 [MCF-7] 细胞 ,  MDA-MB-468 细胞 ,  HCC1937 细胞 ,  MDA-MB-453 细胞 ,  4T1 细胞 ,  MCF 10A 细胞 ,  Annexin V-FITC/ PI荧光双染细胞凋亡检测试剂盒

• Prognosis stratification of patients with breast invasive carcinoma based on cysteine metabolism-disulfidptosis affinity (2023-07-08)

  作者：Yuting Zhang:1.Department of Breast Surgery, The First Affiliated Hospital, Jinan University, Guangzhou, 510000, China. ; Yinghui Ye:1.Department of Laboratory Medicine, Peking University Shenzhen Hospital, Shenzhen, 518000, China. ; Anping Xu:1.Department of Laboratory Medicine, Peking University Shenzhen Hospital, Shenzhen, 518000, China. ; Yulou Luo:1.Department of Breast Surgery, Affiliated Tumor Hospital of Xinjiang Medical University, Urumqi, 830000, China. ; Yutian Sun:1.Department of Medical Oncology, School of Medicine, Sichuan Cancer Hospital and Institute, Sichuan Cancer Center, University of Electronic Science and Technology of China, Chengdu, 610000, China. ; Wei Zhang:1.Department of Breast Surgery, The First Affiliated Hospital, Jinan University, Guangzhou, 510000, China. zhangwei@jnu.edu.cn. ; Ling Ji:1.Department of Laboratory Medicine, Peking University Shenzhen Hospital, Shenzhen, 518000, China. 1120303921@qq.com. ;

  期刊：Prognosis stratification of patients with breast invasive carcinoma based on cysteine metabolism-disulfidptosis affinity

  DOI：10.1007/s00432-023-05028-y

  影响因子 ：3.6

  引用产品： MCF7 [MCF-7] 细胞 ,  MCF7 [MCF-7]细胞专用培养基 ,  BT-20 细胞 ,  BT-20细胞专用培养基 ,  MDA-MB-231 细胞 ,  MDA-MB-231细胞专用培养基 (L15) ,  MDA-kb2 细胞 ,  MDA-kb2细胞专用培养基 (L15) ,  HCC1937 细胞 ,  HCC1937细胞专用培养基

• Scoparone inhibits breast cancer cell viability through the NF‑κB signaling pathway (2023-05-17)

  作者：Xiaoying Wu:1.Mongolian Medical College, Inner Mongolia University for Nationalities, Tongliao, Inner Mongolia Autonomous Region 028000, P.R. China. ; Xiaobo Li:1.Department of Hematology, Mongolian Medical Hospital, Fuxin Mongolian Autonomous County 123199, P.R. China. ; Jing Li:1.Department of Hematology, Mongolian Medical Hospital, Fuxin Mongolian Autonomous County 123199, P.R. China. ; Xinrui Zhao:1.Liaoning Boaotaihe Health Technology Co., Ltd., Fuxin, Liaoning 123000, P.R. China. ; Yongyuan Cui:1.Liaoning Boaotaihe Health Technology Co., Ltd., Fuxin, Liaoning 123000, P.R. China. ; Chaolu Eerdun:1.Medical Department, Inner Mongolia University for Nationalities, Tongliao, Inner Mongolia Autonomous Region 028000, P.R. China. ;

  期刊：Scoparone inhibits breast cancer cell viability through the NF‑κB signaling pathway

  DOI：10.3892/etm.2023.12027

  影响因子 ：2.7

  引用产品： MCF 10A 细胞 ,  MCF7 [MCF-7] 细胞 ,  MDA-MB-231 细胞 ,  ZR-75-1 细胞 ,  HCC1937 细胞 ,  293 [HEK-293] 细胞

• RAMP2-AS1 inhibits CXCL11 expression to suppress malignant phenotype of breast cancer by recruiting DNMT1 and DNMT3B (2004-02-20)

  作者：Li Li:1.Department of Breast Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang 330006, Jiangxi Province, PR China. Electronic address: 156333039@qq.com. ; Ya-Ping Gan:1.Department of Gynaecology and Obstetrics, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou 510120, Guangdong Province, PR China. ; Hui Peng:1.Nanchang University, Nanchang 330006, Jiangxi Province, PR China. ;

  期刊：RAMP2-AS1 inhibits CXCL11 expression to suppress malignant phenotype of breast cancer by recruiting DNMT1 and DNMT3B

  DOI：10.1016/j.yexcr.2022.113139

  影响因子 ：3.7

  引用产品： HCC1937 细胞 ,  MDA-MB-231 细胞 ,  MDA-MB-453 细胞 ,  MCF7 [MCF-7] 细胞 ,  MCF 10A 细胞

• LncRNA OTUD6B-AS1 promotes paclitaxel resistance in triple negative breast cancer by regulation of miR-26a-5p/MTDH pathway-mediated autophagy and genomic instability (2011-01-20)

  作者：Peng-Ping Li:1.Department of Breast-Thyroid Surgery, Department of General Surgery, The First Hospital of Xiaoshan District, Hangzhou, Zhejiang Province 311000, China. ; Rong-Guo Li:1.Department of Breast-Thyroid Surgery, Department of General Surgery, The First Hospital of Xiaoshan District, Hangzhou, Zhejiang Province 311000, China. ; Yu-Qing Huang:1.Department of Breast-Thyroid Surgery, Department of General Surgery, The First Hospital of Xiaoshan District, Hangzhou, Zhejiang Province 311000, China. ; Jin-Pian Lu:1.The Second Affiliated Hospital of Zhejiang Chinese Medical University, Hangzhou, Zhejiang Province 310000, China. ; Wei-Jun Zhang:1.Department of Breast-Thyroid Surgery, Department of General Surgery, The First Hospital of Xiaoshan District, Hangzhou, Zhejiang Province 311000, China. ; Zhen-Yu Wang:1.Department of Breast-Thyroid Surgery, Department of General Surgery, The First Hospital of Xiaoshan District, Hangzhou, Zhejiang Province 311000, China. ;

  期刊：LncRNA OTUD6B-AS1 promotes paclitaxel resistance in triple negative breast cancer by regulation of miR-26a-5p/MTDH pathway-mediated autophagy and genomic instability

  DOI：10.18632/aging.203672

  影响因子 ：5.2

  引用产品： MDA-MB-231 细胞 ,  HCC1937 细胞

• Combination of the 6-thioguanine and disulfiram/Cu synergistically inhibits proliferation of triple-negative breast cancer cells by enhancing DNA damage and disrupting DNA damage checkpoint (2011-01-20)

  作者：Meiran Chu:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun 130021, China; College of Veterinary Medicine, Jilin University, Changchun, Jilin 130062, China. ; Xinglan An:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun 130021, China. ; Daoyu Zhang:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun 130021, China. ; Qi Li:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun 130021, China. ; Xiangpeng Dai:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun 130021, China. ; Hao Yu:1.College of Animal Science, Jilin University, Changchun, Jilin 130062, China. ; Ziyi Li:1.Key Laboratory of Organ Regene

  期刊：Combination of the 6-thioguanine and disulfiram/Cu synergistically inhibits proliferation of triple-negative breast cancer cells by enhancing DNA damage and disrupting DNA damage checkpoint

  DOI：10.1016/j.bbamcr.2021.119169

  影响因子 ：5.1

  引用产品： HCC1937 细胞

• Thioguanine Induces Apoptosis in Triple-Negative Breast Cancer by Regulating PI3K–AKT Pathway (2010-00-20)

  作者：Daoyu Zhang:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun, China. ; Xinglan An:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun, China. ; Qi Li:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun, China. ; Xiaxia Man:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun, China. ; Meiran Chu:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun, China. ; Hao Li:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First Hospital, Jilin University, Changchun, China. ; Nan Zhang:1.Key Laboratory of Organ Regeneration and Transplantation of Ministry of Education, First H

  期刊：Thioguanine Induces Apoptosis in Triple-Negative Breast Cancer by Regulating PI3K–AKT Pathway

  DOI：10.3389/fonc.2020.524922

  影响因子 ：4.7

  引用产品： HCC1937 细胞

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